Varicella zoster infection (ophthalmic zoster)
An ophthalmic zoster is caused by the reactivation of varicella zoster virus belonging to the herpesvirus group (Gürtelrose- herpes zoster). The symptoms and discomfort as a dermatomal rash on the forehead and a painful inflammation of all tissues in the front and rear of rare eye structures can be very intense. Diagnosis is based on the characteristic findings of the anterior structures, with a herpetic dermatitis of the trigeminal nerve 1 (V1) is associated. Treatment consists of oral antiviral drugs, mydriatics and local corticosteroids.
A herpes zoster in the end region is associated in three-quarters of cases with ocular involvement when the nasociliary nerve is affected (due to a lesion at the tip of the nose visible), and in a third of cases without involvement of the nose. Overall, the eyeball is affected in half of the patients.
An ophthalmic zoster is caused by the reactivation of varicella zoster virus belonging to the herpesvirus group (Gürtelrose- herpes zoster). The symptoms and discomfort as a dermatomal rash on the forehead and a painful inflammation of all tissues in the front and rear of rare eye structures can be very intense. Diagnosis is based on the characteristic findings of the anterior structures, with a herpetic dermatitis of the trigeminal nerve 1 (V1) is associated. Treatment consists of oral antiviral drugs, mydriatics and local corticosteroids. A herpes zoster in the end region is associated in three-quarters of cases with ocular involvement when the nasociliary nerve is affected (due to a lesion at the tip of the nose visible), and in a third of cases without involvement of the nose. Overall, the eyeball is affected in half of the patients. A prodromal symptoms and complaints with tingling of the face may occur. During the acute disease, the symptoms and complaints that occur in addition to the painful forehead rash, severe eye pain, eyelid edema pronounced, conjunctival, episcleral and conjunctival hyperemia perikorneale, corneal edema and photophobia may include. Complications keratitis and / or uveitis (overview of uveitis) may be severe and lead to scarring. After-effects-glaucoma, cataracts, chronic or recurrent uveitis, corneal scarring, corneal neovascularization and hypoesthesia-are common and can threaten vision. Postherpetic neuralgia may develop late. Patients can episcleritis (episcleritis) and / or retinitis develop. Herpes Zoster (V1 distribution of the trigeminal nerve) © Springer Science + Business Media var model = {thumbnailUrl: ‘/-/media/manual/professional/images/486-herpes-zoster-v1-slide-32-springer-high_de.jpg ? lang = en & thn = 0 & mw = 350 ‘, imageUrl:’ /-/media/manual/professional/images/486-herpes-zoster-v1-slide-32-springer-high_de.jpg?la=de&thn=0 ‘, title ‘herpes zoster (V1 distribution of the trigeminal nerve),’ description ‘ u003Ca id = “v37894232 ” class = “”anchor “” u003e u003c / a u003e u003cdiv class = “”para “” u003e u003cp u003eBei this patient with acute herpes zoster developed vesicular lesions on the V1 distribution of the ophthalmic branch of the trigeminal nerve. Incorporating their nose also points to the involvement of the nasociliary nerve that supplies the cornea u003c / p u003e u003c / div u003e ‘credits’. © Springer Science + Business Media’