Locked-In Syndrome

The locked-in syndrome is a condition of alertness and awareness associated with quadriplegia and paralysis of the lower cranial nerve, which is the inability of the invention to show a facial expression, to move, to speak, or, with the exception of encoded eye movements to communicate.

A locked-in syndrome is typically the result of a pontine hemorrhage or infarction, causing quadriplegia and the lower cranial nerves and the centers that control the horizontal eye movements, suspend and destroyed. Less common causes include other diseases that cause severe extensive motor paralysis (z. B. Guillain-Barre syndrome), and cancers involving the posterior fossa and the pons.

The locked-in syndrome is a condition of alertness and awareness associated with quadriplegia and paralysis of the lower cranial nerve, which is the inability of the invention to show a facial expression, to move, to speak, or, with the exception of encoded eye movements to communicate. A locked-in syndrome is typically the result of a pontine hemorrhage or infarction, causing quadriplegia and the lower cranial nerves and the centers that control the horizontal eye movements, suspend and destroyed. Less common causes include other diseases that cause severe extensive motor paralysis (z. B. Guillain-Barre syndrome), and cancers involving the posterior fossa and the pons. Patients have an intact cognitive function and are awake, with eyes open and normal sleep-wake cycles. You can hear and see. However, they can move neither its lower face, nor can they chew, swallow, speak, breathe, move their arms and legs or run sideways eye movements. A vertical movement of the eyes is possible; the patient can open and close her eyes or blink a certain number to answer questions. Diagnosis Clinical Evaluation The diagnosis is primarily clinical. Because the patient’s motor responses (eg. As retreating back to painful stimuli) are missing, which are typically used to measure the reactivity, they can be mistaken for unconscious. Therefore, it should in all patients who can not move, the understanding will be tested by the invitation, squinting his eyes or perform vertical eye movements. Like the vegetative Zutsand a neuroradiological imaging is indicated to rule out treatable diseases. Imaging of the THE BRAIN with CT or MRI is performed, and helps to identify the pontine anomaly. PET, SPECT, or functional MRI can be performed for further evaluation of brain function if the diagnosis is in doubt. In patients with locked-in syndrome, the EEG shows normal sleep-wake pattern. Prognosis The prognosis depends on the cause, and the subsequent level of support that is provided. A locked-in syndrome due to transient ischemia or stroke in a small-vertebrobasilaris A. area may be, for. B. completely regress. Partially reversibeler cause (z. B. Guillain-Barre syndrome) can occur for months a recovery, but it is rarely complete. Favorable prognostic features are Early recovery of lateral eye movements An early recovery from evoked potentials in response to magnetic stimulation of the motor cortex Irreversible or progressive disorders (eg., Cancer diseases involving the posterior fossa and the pons) are usually fatal. Treatment Supportive treatment of supportive treatment include the mainstay of therapy for patients with locked-in syndrome and should include prevention of systemic complications due to immobilization supply of good nutrition prevention of pressure ulcers Providing (such as pneumonia, urinary tract infection, thromboembolism.) physical therapy for contracture There is no specific treatment. Speech therapists can help to establish a communication code using eye blinks or eye movements. Since cognitive function intact and communication is possible, patients should make their own decisions about health care. Some patients with locked-in syndrome to communicate over the Internet using a computer terminal that is controlled by eye movements and other aids.

Health Life Media Team

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