Cor Pulmonary

When pulmonary heart a right enlargement occurs due to a lung disease that causes pulmonary arterial hypertension. The result is the right ventricular failure. Clinical findings include peripheral edema, jugular veins congestion, hepatomegaly and parasternales lifting. The diagnosis is made clinically and by echocardiography. The treatment depends on the particular cause.

The pulmonary heart disease is caused by a disease of the lungs or their blood vessels. It does not describe the RV-LV-magnification secondary to failure to a congenital heart disease (z. B. ventricular septal defect) or acquired valve disease. The pulmonary heart disease is usually chronic, but can also be acute and reversible. The primary pulmonary hypertension (i. E. Not caused by pulmonary or cardiac disease) is discussed elsewhere (pulmonary hypertension).

When pulmonary heart a right enlargement occurs due to a lung disease that causes pulmonary arterial hypertension. The result is the right ventricular failure. Clinical findings include peripheral edema, jugular veins congestion, hepatomegaly and parasternales lifting. The diagnosis is made clinically and by echocardiography. The treatment depends on the particular cause. The pulmonary heart disease is caused by a disease of the lungs or their blood vessels. It does not describe the RV-LV-magnification secondary to failure to a congenital heart disease (z. B. ventricular septal defect) or acquired valve disease. The pulmonary heart disease is usually chronic, but can also be acute and reversible. The primary pulmonary hypertension (i. E. Not caused by pulmonary or cardiac disease) is discussed elsewhere (pulmonary hypertension). Pathophysiology lung diseases cause pulmonary hypertension by different mechanisms: loss of capillaries (. Eg due vesicular changes in COPD or thrombosis in pulmonary embolism) vasoconstriction by hypoxia, hypercapnia, or both Elevated alveolar pressure (for example, in COPD, during mechanical ventilation.) Medial hypertrophy in the arterioles (often a reaction to the pulmonary hypertension due to other mechanisms) pulmonary hypertension increases afterload of the RV, which triggers a cascade of events similar to an LV failure, incl. increased end-diastolic and central venous pressure, ventricular hypertrophy and dilatation. The requirements for the RV may increase by increased blood viscosity due to hypoxia-induced polycythemia. Rarely betriff an RV failure and the LV, if a dysfunctional septum into the LV bulges, interferes with the filling of the LV and causes in this way diastolic dysfunction. Etiology Acute pulmonary heart disease has few causes. Chronic pulmonary heart disease is usually caused by COPD, but there are also some less common causes (see Table: Causes of cor pulmonale). In patients with COPD acute exacerbation or pulmonary infection can trigger the RV overload. In chronic pulmonary heart disease, the risk of venous thromboembolism is increased. Causes of Cor Pulmonary occurrence cause of acute massive pulmonary embolization injury from mechanical ventilation (most common in ARDS) * Extensive Chronic COPD loss of lung tissue by surgery or trauma Chronic, unresolved pulmonary embolism pulmonary veno-occlusive disorders Systemic sclerosis Pulmonary interstitial fibrosis kyphoskoliosis obesity with alveolar hypoventilation Neuromuscular Diseases respiratory muscle idiopathic alveolar hypotension * COPD is the leading cause of chronic pulmonary heart disease. ARDS = acute respiratory distress syndrome. First, the symptoms and complaints pulmonary heart disease is asymptomatic, although patients usually have significant symptoms (eg. As dyspnea, fatigue on exertion) due to underlying lung disease. Later, when the RV pressure increases, among the physical signs often a systolic parasternales lifting, a noisy component of the pulmonary second heart sound (S2) and noise of the functional tricuspid – and Pulmonalklappeninsuffizienz. Still later, a right ventricular gallop rhythm can (3. [S3] and fourth [S4] Heartbeat) reinforced during inspiration, extended jugular veins (with a dominant a-wave, if not a tricuspid regurgitation is present), hepatomegaly and edema of the lower extremity occur. Diagnosis Clinical suspicion echocardiography A pulmonary heart disease should be suspected in all patients who have any of the above causes. The chest X-ray photograph shows the increase in the RV and the proximal Pulmonalarteriensegmente with distal arterial dilution. ECG sign of RV hypertrophy (z. B. rightward shift of the axis of the heart, QR wave in lead V1 and the R wave dominant in leads V1 to V3) correlate well with the extent of pulmonary arterial hypertension. Since the pulmonary hyperinflation and bullae in COPD cause a change in position of the heart, the physical examination, the X-ray and ECG may not be groundbreaking. The echocardiography or scintigraphy is performed to determine the LV and RV function, echocardiography can determine the systolic RV pressure, but is often limited in the technical application by the lung disease. A cardiac MRI may be helpful to evaluate the cardiac chambers and function in some patients. A right heart Kather investigation may be needed to confirm the findings. Cor Pulmonary (echocardiogram) © Springer Science + Business Media var model = {thumbnailUrl: ‘/-/media/manual/professional/images/525-cor-pulmonale-echocardiogram-s118-springer-high_de.jpg?la=de&thn=0&mw = 350 ‘, imageUrl’ /-/media/manual/professional/images/525-cor-pulmonale-echocardiogram-s118-springer-high_de.jpg?la=de&thn=0 ‘, title:’ cor pulmonale (echocardiogram) ‘ , description: ‘ u003Ca id = “v38395841 ” class = “”anchor “” u003e u003c / a u003e u003cdiv class = “”para “” u003e u003cp u003eKurzachsenansicht

Health Life Media Team

Leave a Reply